Transcript
Respiratory Update II
Deborah A. Hood DNP RN-BC CM
Course Objectives:
Describe risk factors, treatments and nursing care of the patient with obstructive sleep apnea.
Describe risk factors, treatment and nursing care of the patient with head and neck cancer.
Describe risk factors, treatments, and nursing care of the patient with COPD, pneumonia, and influenza.
Describe the care and management of the critically ill patient suffering from pulmonary embolus.
Disorders of the Oral Pharynx and Tonsils (Non-Infectious)
Obstructive Sleep Apnea (OSA)
Head and Neck Cancer
Obstructive Sleep Apnea (OSA)
Cessation of breathing during sleep lasting longer than 10 seconds at a minimum of 5 times per hour.
Cause maybe neurogenic, but is usually due to problems with the soft palate or tongue.
Contributing factors are obesity, a large uvula, a short neck, smoking, enlarged tonsils/adenoids, and oropharyngeal edema.
Men are affected more than women.
Obstructive Sleep Apnea
History
Do you wake up tired?
Do you experience day time sleepiness?
Are you more irritable?
Has anyone complained of personality changes?
Does your significant other report snoring or periods where you stop breathing?
Obstructive Sleep Apnea (OSA)
Pathophysiology:
Muscles relax during sleep, displacing the tongue and neck structures causing obstruction of the upper airway though the chest continue to move.
Apnea causes a respiratory acidosis.
Neural centers of brain are stimulated after 10 seconds or longer of apnea and then corrects the obstruction.
Obstructive Sleep Apnea (OSA)
Obstructive Sleep Apnea (OSA)
Physical Assessment
Observe during sleep
Decreased pulse ox
Cyanosis
Cyclical cessation of breathing
Snoring
Obstructive Sleep Apnea (OSA)
Diagnostics: Overnight sleep studies
Polysomnography (PSG) –overnight study
Electroencephalograph (EEG)
Electrocardiograph (ECG)
Electromyograph (EMG)-determines depth of sleep, respiratory effect, 02 saturation, muscle movement
Pulse oximetry
Obstructive Sleep Apnea
Obstructive Sleep Apnea (OSA)
Non-surgical Management
Change in sleeping position
Weight loss
Position-fixing devices
Positive-pressure ventilation (CPAP)
Surgical Management
Adenoidectomy
Uvulectomy
Remodeling of posterior oropharynx
Obstructive Sleep Apnea
Head and Neck Cancers
Accounts for about 4% of carcinomas in the U.S
Men are affected three times more than women.
Occur in people 60 yrs or older, but becoming more prevalent in young adults.
Risk factors:
Tobacco abuse
Alcohol abuse
Voice abuse (chronic laryngitis)
Chronic exposure to chemicals/pollutants
Poor oral hygiene
Long-term or severe GERD
Head and Neck Cancer
Pathophysiology
80% are squamous cell carcinomas—slow growing. Usually first appear as deep ulcerations.
Begins with a chronically irritated mucosa that changes into a tougher mucosa (squamous metaplasia).
It is then replaced by an increasing tougher, thicker mucosa (hyperplasia).
Keratin layer develops (keratosis).
Genes controlling cell growth is damaged leading to abnormal, malignant cells. These lesions are often white and patchy or red and velvety.
Metastasis often occur to lymph nodes, muscle, bone, or systemically through blood and lymphatic systems.
Head and Neck Cancers
History
Have you had any difficulty in speaking? Do you currently or have you ever abused alcohol or tobacco?
Exposure to environmental/occupational pollutants.
Have you noticed and sores or lump in your neck?
Do you have chronic lung disease?
Head and Neck Cancers
Physical Assessment:
Inspection and palpation of the head and neck to assess for lesions, lumps, and tumors.
Laryngeal exam by independent practitioner
Common Diagnostics:
Labs: CBC, Coagulation Studies, UA, Blood chemistries (liver, renal).
CT to evaluate tumor location
MRI to differentiate normal from diseased tissue (evaluate soft-tissue invasion).
SPECT/PET: to locate additional tumor sites.
Laryngoscopy, esophagoscopy, bronchoscopy.
Head and Neck Cancers
Head and Neck Cancer
Head and Neck Cancers
Head and Neck Cancers
Head and Neck Cancers
Head and Neck Cancers
Treatment:
Non-surgical
Radiation
Chemotherapy
Surgical
Small tumors maybe removed with laser/photodynamic therapy.
Laryngectomy (partial/total)
Tracheostomy
Oropharyngeal cancer resections
Head and Neck Cancers
Post-op Care
Monitor airway patency
Suctioning
Coughing and deep breathing
Adequate oxygenation
Humidified oxygen
Monitor vital signs
Hemodynamic status
Comfort level
Provide a means for communicating
Head and Neck Cancers
Post-op care:
Monitor for hemorrhage
Empty drains. Keep clear of clots by “stripping/milking” if there is an MD order.
Document in output.
Wound care/stoma care
Pain management
Emotional support
Ensure consults:
SLP
Nutritional services
OT/PT
Lower Respiratory Problems
Chronic Airflow Limitations
Asthma-intermittent with obstructive air flow and wheezing - REVERSIBLE
COPD – causes irreversible tissue damage and leads to respiratory failure
Chronic bronchitis
Emphysema
Asthma
Can occur at any age. Most adults with asthma had it as a child.
More common in urban settings than rural.
ASA or NSAID’s may trigger asthma.
More than 40 million Americans suffer from asthma.
5000 deaths from acute asthma occur in the U.S annually.
Asthma
Pathophysiology:
Intermittent and reversible airflow obstruction affecting only the airways, due to:
Inflammation obstructs the lumen of the airways. Allergens bind to antibodies (IgE). The inflammatory response begins in which histamine, mast and WBC cells congest the area. Blood vessels dilate, capillaries leak leading to more swelling and congestion due to mucus production.
Bronchospasm: airway hyper-responsiveness (allergens/pollutants) causes narrowing due to constriction. Severe bronchospasm can limit airflow to alveoli.
Asthma
Asthma
History:
How often do you have attacks?
What are your triggers?
Recent URI?
Experienced other allergic symptoms (skin, rhinitis)
History of smoking?
Asthma
Physical Assessment:
Audible wheeze (louder on exhalation)
Increased respiratory rate
Coughing
Accessory muscle use
“barrel chest”
Unable to speak in complete sentences
Hypoxemia
Changes in LOC
Asthma
Common Diagnostics:
Labs: ABG’s, CBC, drug levels (Theophylline)
Pulmonary Function Tests
Chest X-ray
Asthma
Asthma
Treatment:
Drug therapy
Preventative Medications: decrease airway responsiveness.
Bronchodilators (long acting: Serevent)
Cholinergic antagonist (Atrovent)
Corticosteroids (Flovent, Prednisone)
NSAID (Tilade)
Methylxanthines (Theo-dur)
Leukotriene Antagonist (Singulair, Xolair)
Rescue Drugs: stop attack once it starts
Bronchodilators (short-acting: Albuterol, Xopenex)
Cholinergic antagonist
Asthma
Patient/Family Teaching
Goals to improve airflow by decreasing the frequency of attacks.
Education
Assess respiratory status with peak flow at lease twice per week.
How to self-treat
When to consult practitioner
Drug therapy
Compliance
Use spacer
Lifestyle management
Exercise (aerobic)—may require pre-medication
Cessation of smoking
Oxygen therapy
Minimize exposure to allergens
Asthma
COPD
Chronic Bronchitis
Emphysema
COPD
Prevalence of chronic bronchitis and emphysema is about 13.5 million (chronic bronchitis) and 2 million (emphysema).
Fourth leading cause of morbidity and mortality in the U.S.
Chronic Bronchitis
Pathophysiology:
Inflammation of the bronchi and bronchioles caused by chronic exposure to irritants.
Chronic inflammation increases the number and size of mucus glands thus more mucus is produced.
Bronchial walls thicken and impair airflow.
Mucus plugs and infection narrow airways causing decreased gas exchange.
Emphysema
Pathophysiology:
Two major changes occur:
Loss of lung elasticity
Hyperinflation of the lung
COPD
History:
Occupational history
Smoking history
What are the patients triggers?
Assess cough pattern
Has sputum production changed? When do you experience the most sputum?
Problems with ADL’s, sexual activity, or sleeping?
Experienced unplanned weight loss?
COPD
Physical Assessment:
General appearance
Rapid, shallow breathing
Barrel chest
Fremitus
Hyperresonant sounds on percussion
Wheezes on inspiration and expiration
Delayed capillary refill
Pallor/Cyanosis
Clubbing to the digits
Lower extremity edema (right sided heart failure)
COPD
Common Diagnostics:
Labs: ABG’s, CBC, sputum for gram stain and C&S,
Chest X-ray
PFT’s
COPD
COPD
Treatment:
Non-surgical management:
Airway maintenance (liquefy secretions)
Cough enhancement
Oxygen therapy
Drug therapy
Pulmonary rehabilitation
Exercise conditioning
Breathing exercises
Surgical management:
Lung reduction
Lung transplantation
COPD
COPD
Patient/family teaching:
Disease teaching
Avoidance of infectious agents
Medication instruction
Nutrition regimen
Exercise regimen
Oxygen precautions
Smoking cessation
Infectious Respiratory Problems
Influenza
Pneumonia
Influenza
Highly contagious acute viral respiratory infection.
Patients are contagious 24 hrs prior to becoming symptomatic.
Can lead to complications (pneumonia, respiratory failure, death).
Annual vaccinations are recommended for all, especially for the elderly as well as those who are immunocompromised, or debilitated.
Teach patients to wash hands often and observe the new “etiquette” surrounding coughing and sneezing. They should isolate themselves until completely recovered.
Influenza
Physical Assessment:
Severe headache
Muscles aches
Fever
Chills
Fatigue
Weakness
Anorexia
Sore throat
Rhinorrhea
Pneumonia
2-5 million cases annually.
Seventh leading cause of death.
Highest incidence among institutionalized older adults.
Maybe community or hospital acquired. Community is the most common.
Pneumonia
Pneumonia
Pathophysiology:
Excess of fluid in the lungs due to the inflammatory process that is triggered by inhalation of infectious organisms or irritating agents.
Inflammation occurs in the interstitial spaces, the alveoli, and often the bronchioles due to the invasion of the bacteria causing edema and formation of exudate.
Alveoli walls thicken impeding oxygenation and perfusion.
Fibrin and edema stiffen the lung reducing compliance and vital capacity.
Pneumonia may start in a segment then progress to a lobe.
May lead to septicemia if organisms move into the bloodstream.
Pneumonia
History:
Assess risk factors:
Older adult
Did not receive a recent immunization for pneumonia.
Chronic health problems/chronic lung disease
Tobacco/alcohol abuse/illicit drugs
Living/working conditions
Diet
Exercise
Sleep patterns
Pneumonia
Physical Assessment:
General appearance
Chest/pleuritic pain
Myalgia
Headache
Chills
Fever
Cough (sputum production)
Respiratory distress
Tachycardia (rapid, weak)
Hypotensive (dehydration and vasodilation)
Crackles, wheezing, bronchial sounds to affected lung field
Pneumonia
Common Diagnostics:
Labs: sputum for gram stain and C&S, CBC (increased or decreased WBC count), BC, UA/UC, HIV testing.
CXR may not become diagnostic for 2 days after onset of symptoms.
Pulse oximetry
Bronchoscopy
Pneumonia
Pneumonia
Treatment
Oxygenation
Airway clearance
Fluid resuscitation
Medications:
Antibiotic therapy
Bronchodilators
Steroids
Respiratory Problems of the Critically Ill
Pulmonary Embolism
Pulmonary Embolism
Can occur at any age.
Most common pulmonary disease among hospitalized patients.
Major risk factors for DVT leading to PE:
Prolonged immobility
Central venous catheters
Surgery
Obesity
Advancing age
Conditions that increase blood clotting
History of thromboembolism
Pulmonary Embolism
Patient History:
Assess sudden onset of distress
Determine risk factors
Does the patient have a history of DVT, recent surgery, or prolonged immobility?
Pulmonary Embolism
Pathophysiology:
Collection of particulate matter (solid, liquid, air) that enters venous circulation (vena cava to right atrium) and lodges in a pulmonary artery. Platelets collect on the embolus causing vasoconstriction which leads to pulmonary hypertension.
Blood flow is obstructed leading to reduced oxygenation of the body.
Pulmonary Embolism
Sources of emboli:
Fat
Oil
Tumor cells
Amniotic fluid
Foreign objects (broken IV catheters)
Injected particles
Septic emboli
Pulmonary Embolism
Physical Assessment:
Symptoms:
Sudden onset of dyspnea
Pleuritic chest pain
Described as sharp, stabbing on inspiration
Severe apprehension, restlessness
Feeling of impending doom
Cough
Hemoptysis secondary to pulmonary infarction
Dry
Pulmonary Embolism
Physical Assessment:
Tachycardia
Breath sounds maybe normal or they may have crackles
Pleural friction rub
Tachypnea
Diaphoresis
Low-grade temp
Petechiae over chest and axilla
Decreased oxygen saturation and PaO2
S3 and S4 heart sound
Severe hypotension
Pulmonary Embolism
Common Diagnostics:
ABG’s
Spiral CT
Chest X-ray (infiltrates)
Venous doppler
Transesophageal echocardiogram (TEE)
VQ Lung Scan
Pulmonary Embolism
Pulmonary Embolism
Pulmonary Embolism
Treatment: Medical
Oxygen
Drug Therapy
Anticoagulants
Lovenox
Heparin drip
Coumadin therapy
Fibrinolytics
Retavase
TPA
Pulmonary Embolism
Treatment: Surgical
Embolectomy: surgical removal of the embolus from the pulmonary blood vessel.
Inferior vena cava interruption/filter
Pulmonary Embolism
Inferior Vena Cava Filter
Pulmonary Embolism
Pulmonary Embolism
Nursing management/teaching:
Anxiety
Support family/patient
Monitor for bleeding
Advise patient/family on bleeding precautions
Decreased Cardiac Output
Inotropic support
IV fluid therapy
Manage hypoxemia
Pulmonary Embolism
Prevention:
Avoid prolongation of bed rest
Assess risk factors and intervene
DVT prophylaxis
Teach patient to adhere to Coumadin therapy
SCD’s/venodynes
SQ heparin/low-molecular weight heparin (Lovenox)
References
Ahrens, T., Prentice, D., & Kleinpell, R. (2007). Pulmonary Anatomy and Physiology. In Quincy McDonald (Ed.), Critical Care Nursing Certification (5 ed., pp. 171-227). New York: McGraw-Hill.
Ignatavicius, D. D., & Workman, L. M. (2010). Medical-Surgical Nursing Patient-Centered Collaborative Care (6 ed.). St. Louis: Saunders Elsevier.
References (Images)
www.anaesthetist.com
www.clivir.com
www.diagnosingsleepapnea.com
www.dkimages.com
www.examiner.com
www.fn.bmj.com
www.images.timnhanh.com
www.metrohealth.org
References (Images)
www.powershowz.medicalillustration.com
www.thesleepapneaonline.com
www.aboutneckcancer.com
www.riversideonline.com
www.surgery.med.umich.edu
www.uth.tmc.edu
www.news.wustl.edu/news