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a. What is one possible double stranded DNA sequence you would insert into this gene is order to have it now make a protein with the 1o structure: NH2 MET ALA SER SER ILE VAL GLU COOH
b. Which bases in the default wildtype sequence (from # to #) would you replace with this new stretch of sequence?
Male sterility in maize is determined by which type of mutation (in this case)?
Hi, I was uncertain about what would be the correct alternative on a question I have to answer, regarding male sterility in maize. Below, I’ll insert the problem and what I have thought so far (also, my apologies if the sentence is not well formulated, I’m translating it from my native language).
QUESTION In maize, a plant has both male and female flowers. Male steri
Which of the following statements about the prevalence of neurological disease in the U.S. today is
Which of the following statements about the prevalence of neurological disease in the U.S. today is false?
a. There are more people diagnosed with stroke than with epilepsy. b. There are more people diagnosed with Alzheimer's disease than with head trauma. c. There are more people diagnosed with cerebral palsy than with epilepsy. d. There are more peop
Use the following hypothetical information to answer the next two questions. Eye colour in humans appears to be controlled by multiple alleles. The green allele and the brown allele are dominant to blue. Green eye colour is expressed only in the absence of a brown allele. Red-green colour blindness is a sex-linked trait. Red-green colour blind
A black body color (as opposed to the wild-type yellow color) in fruit flies is due to the recessive ebony allele, e. Small wings are due to the recessive vestigal allele, vg. You wish to deduce the genotype of a yellow-bodied large-winged male fruit fly. What are all the possible genotypes this male could be at the ebony and vestigal loci? What is the genotype an
Complementation problem: possible region of deletion place the mutation on the thick line which ...
I need help with part B of this problem, I've asked Genetics tutors, other professors and even went on chegg to use the expert Q&A -- I've done part A and I know that 3 complements 1, 2 and 6 are unable to complement and 3,4,5 and 1,2 6 have similar mutations
but I have no idea if I am right? my genetics professor says that if I look at row 4 and
1. The first child of this couple was Rh- (Rhesus negative). What is the chance that the second child of this is a girl and Rh- (Rhesus negative)? Use Punnett squares and/or the calculation to show your work.
2. Nomenclature for writing alleles on a homologous chromosome pair
The phrase DEP/dep means the first three letters, DEP, represent the order in
Though we often think of genes in terms of the phenotypes they produce.......
Though weoften think of genes in terms of the phenotypes they produce (e.g., curly leaves, flaky tail, brown eyes), the molecular function of most genes is to encode proteins. Many cellular proteins function as enzymes. The table that follows describes the map distance between six different genes that encode six different enzymes: Ada, adenosine deaminase, Hao-1, hydroxy
What is the map distance to 4 significant figures between the A and B loci?
"These are the results of a three point test cross in Drosophila for the three loci ABC: aCB = 1444, ACb = 245, AcB = 1222, ACB = 2212, aCb = 1333, acB = 299, Acb = 1499, acb = 2333. What is the map distance to 4 significant figures between the A and B loci? Hint: Establish gene order first."
What is the map distance to 4 significant figures between A and B loci?
"These are the results of a three point test cross in Drosophila for the three loci ABC: aCB = 354, ACb = 28, AcB = 261, ACB = 666, aCb = 256, acB = 35, Acb = 309, acb = 578. What is the map distance to 4 significant figures between the A and B loci? Hint: Establish gene order first."
How can I determine how many Genetic Loci control a specific trait?
So I have a question about finding how many loci control a certain trait. I don't know how to determine it.
The info: (Disregard any outside info retaining to the specific animal)
Pure-bred male White Cat mated with a Pure-bred female Brown Cat. They produced 90 progeny ALL Brown. Randomly interbreeding F1, they produced 1600 F2 progeny. 9
1. 12 out of 2400 babies that were born at a maternity hospital died shortly after birth from colonic obstruction due to a recessive allele. Determine the frequency of the lethal allele in the population, expressed as a percent.
2. 10 of 2400 babies born at a maternity hospital died shortly after birth from colonic obstruction due to a recessive allele. What percent
Cell-based manufacturing in animal cell tissue culture
I am interested to gain more knowledge on this topic. I be really grateful if you guys can provide any information regarding this topic (books, journals...etc). Your feedback is highly appreciated.
The first two prenancies for a couple result in late term stillbirths...
The first two pregnancies for a couple result in late term stillbirths. The reproductive histories of both families, over 3 generations, were compiled. A pattern of frequent miscarriages, stillbirths and malformed babies that died shortly after birth were seen in the husband's family. The husband has a karyotype done, which shows that he has the normal number of chromosomes. Wh
Help with Identificatying Restriction Enzyme Recognition Sites in a DNA sequence
Hi Folks,
I'm really struggling with a PCR & restriction enzyme question that's part of my formative (unmarked) work for the summer months in preparation from an Applied Biology course at University.
The entire set of questions are set out on an instruction sheet so I'll try to replicate them here as I'm struggling to fully understand ho
Deletions, insertion, and frameshift mutations are more serious than a point
I got the following questions wrong on a test/assignment. Can someone let me know what the correct answers are so I have the correct information for my final exam. Thx
#1 Deletions, insertions, and frameshift mutations are more serious than a point mutation because A. mis-sense mutations result in more malfunction than nonsense mutations B.&nb
What would be the size in base pairs of the largest and smallest amplicons that would be expected if
Relevant details of the STRs are as follows
TH01
Chromosomal location: 11p15.5 (tyrosine hydroxylase, 1st intron) Repeat motif: TCAT (GenBank top strand) although AATG (bottom strand) often used. Note that variants with a [AATG]nATG[AATG]n structure quite common e.g. [AATG]6ATG[AATG]3 would be 9.3 repeats. Allele range: 3-14
Was Lewis just mapping them on a linkage map based on mutations?
Hi guys,
So I've spent a few days reviewing the genetic tools used in labs, but can't quite find the distinction between a few of the methods.
1. RT-PCR is used to determine if a specific gene is expressed; however, RNA-seq appears to do the same thing. I understand that RT-PCR uses a specific primer and thus tests the expression of one gene. Does RNA
I got my finals in a few days and was wondering if anybody could give me the link to the answers to the review questions in this book? I finished all the questions but I'm not entirely sure how to check them as there is no answer section and the wwnorton.com/studyspace doesn't have anything either. Thanks
Hi there, I was wondering if someone could let me know if I did this right or not. Thanks in advance for any help.
So, the problem states the following:
Quote
We have crossed one flower with a corolla that is white and personé (the original question is not in English and I can't find a definition for personé anywhere, doesn't really matter I
Can someone please tell me what is the formulae for calculating genetic map units?
Here is the question from my quiz:
The cross GE/ge × ge/ge produces the following progeny: GE/ge 404, ge/ge 396, gE/ge 97, Ge/ge 103. From these data, one can conclude that there are 20 map units between the G and E loci.
People with the recessive disease, Purple Tongue Syndrome, are unable to make the enzyme Blue Pigment Decolorase (BPD). You isolate mRNA from a person (P) afflicted with the disease and from a normal person (N) and do the following: i. Mix excess P mRNA with cloned genomic DNA carrying the BPD gene and, separately, mix Normal (N) mRNA with the same DNA. ii. Raise the tempe
Two cats having short tails are mated and have 11 kittens. The offspring (kitte
Two cats having short tails are mated and have 11 kittens. The offspring (kittens) from this cross yield interesting results. Of the 11 kittens, 3 are born with long tails, 2 are born with no tails and 6 are born with short tails. What is the simplest genetic explanation for this litter of kittens and their parents? Provide the genotypes of the parents and al
13) In Drosophila, assume that the gene for scute bristles (s) is located at map position 0.0 and that the gene for ruby eyes (r) is at position 15.0. Both genes are located on the X chromosome and are recessive to their wild-type alleles. A cross is made between scute-bristled females and ruby-eyed males. Phenotypically wild F1 females were then mated to homozygous double mutant ma
Two different genes control the expression of coat colour in an organism.
3. Two different genes control the expression of coat colour in an organism. The allele B produces a black coat colour and the allele G produces a gray-striped coat. The gene B influences the expression of gene G. The gray-stripped coat pattern appears only when gene B is homozygous recessive. An albino (coat colour lacking) occurs when both genes are homozygous. A pure-breedin
Fly larva mutations for Gap, pair-rule, and maternal effect genes
The title say most of it. I'm supposed to draw the mutations in the Drosophila larva, but I also need to label the structures which my textbook is not helping me much with. So far I just know that the mutants are stubby little creatures... I also need to list the specific gene mutated since I guess these are gene families???
Use the following table of progeny phenotypes for 7 different deletions
Deletions can be used to map genes along a chromosome. In order to do this a series of crosses in which one parent is homozygous for a mutant allele is crossed with the other parent that is homozygous for a partial deletion of the region. The progeny are scored to determine whether they have the mutant phenotype ("m") or the wild-type phenotype ("+"). If a mutati
im quite new to NCBI and genebank so heres my question
i need to extract sequences from 7 geneloci of 130 different bacterial strains deposited on genebank. those sequences were all submitted in the context of one single study. now instead of the time consuming process of extracting the sequences strain per strain in the taxonomy browser ,
How many possible alleles are at a locus consisting of 3 base-pairs in a diploid
ugh, I am so confused...
So, I guess a locus is just a spot for a gene, so the gene is also considered to consist of 3 bases? Gene: _ _ _ Each "_" could be either an A,T,C, or G. So there are 4 possibilities per "_" So, there are 4 x 4 x 4 = 64 different possible alleles of a gene on one chromosome...? Since there are 2 c
Please help me answer this question. AaBbCc x aabbcc test cross with ABC linked and dominant. 2000 phenotypes observed with the following individuals counted out of 2000. establish the gene arrangement and map distances.
The likelihood of having identical children (who are not twins)
So I figure if you have enough babies (probably in the billions) you'd eventually get one that was essentially genetically and/or aesthetically identical to another. Could anyone give me any background information and numbers regarding the likelihood of this?
I am in serious need of correction or confirmation please! I am going to throw out some random facts and please let me know if I am accurate or not. Thanks for helping!
F1 hybrid can only be created by using two separate parents from inbred lines or P1. F1 are uniform, showing a new phenotype without variation. Not even once in a while will you see a second phenotype show
Thank you for reading. So I have a basic understanding of Mendel's works, but I am trying to figure genetics out better. Please if you could help, because I can't find the data anywhere on the internet on these questions.
If you take an F1 hybrid and cross it to a p1 (seperate from the hybrid's two different parent types) would you get a new F1 hybrid, or F
What happens if region 1-100 is removed from the Gal4p sequence?
Can someone verify the validity of my statements? (This topic is extremely confusing to me)
A) What happens if region 1-100 is removed from the Gal4p sequence? Gal4p is a protein encoded by the GAL4 gene and the entire protein consists of a total of 881 amino acids. Section 1-98 of the entire Gal4 protein is called the “DNA binding domain.” In order for transc