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BUDDA2222 BUDDA2222
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6 years ago
What are the two major divisions of the nervous system?
 a. endocrine and exocrine nervous systems
  b. upper and lower nervous systems
 c. spinal and extremity nervous systems
 d. central and peripheral nervous systems
  e. cerebral and autonomic nervous systems

Q. 2

Which of the following medical conditions could potentially trigger RBC production?
 a. Cerebrovascular accident
  b. Acute renal failure
  c. Hypoxia
  d. Hepatitis

Q. 3

Respiratory diseases, such as chronic obstructive pulmonary disease, result in inability to maintain adequate oxygenation or release of _______________.
 Fill in the blank(s) with correct word

Q. 4

Define sickle cell anemia and describe the role of nutrition therapy in sickle cell anemia.

Q. 5

Which of the following nutrients helps to maintain the flexibility of the membrane of red blood cells?
 a. Vitamin E
  b. Vitamin K
  c. Vitamin A
  d. Vitamin B12

Q. 6

The _______________ represents the difference between unmeasured anions and cations.
 Fill in the blank(s) with correct word

Q. 7

Discuss how the feedback mechanism for iron absorption is altered in iron-deficiency anemia.
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Replies
wrote...
6 years ago
Answer to #1

d

Answer to #2

c

Answer to #3

carbon dioxide

Answer to #4

Sickle cell anemia is the most common structural hemoglobinopathy, defined as homozygous abnormal hemoglobin polymerization resulting in a sickling of RBCs in symptomatic individuals. The crescent-shaped cell morphology is obvious when stained and magnified. Nutrition therapy for an acute sickle cell crisis should include increased macronutrients because this condition increases energy expenditure, protein and bone turnover, risk of pulmonary disease, chronic inflammation, frequency of infections, and cardiac output.

Answer to #5

a

Answer to #6

anion gap

Answer to #7

Persons with uncomplicated deficiencies of iron already have up-regulated proteins for enhancing iron absorption from the digestive tract. One such protein (hepcidin) communicates iron status and erythropoietic demands to the small intestine and modulates absorption. This feedback mechanism allows deficient individuals to enhance their absorption of available heme and non-heme iron. Iron-deficiency anemia can be corrected through continued iron-dense, nutrient-dense dietary intake, supplementation, and correction of any underlying conditions that may be contributing to the deficit. Increasing the nutrient density of foods over the long term is the best strategy.
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