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alyssaa1017 alyssaa1017
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6 years ago
When adrenaline binds to adrenergic receptors on the surface of a muscle cell, it activates a G protein, initiating an intracellular signaling pathway in which the activated  subunit activates adenylyl cyclase, thereby increasing cAMP levels in the
 
  cell. The cAMP molecules then activate a cAMP-dependent kinase (PKA) that, in turn, activates enzymes that result in the breakdown of muscle glycogen, thus lowering glycogen levels. You obtain muscle cells that are defective in various components of the signaling pathway. Referring to Figure Q16-36, indicate how glycogen levels would be affected in the presence of adrenaline in the following cells. Would they be higher or lower than in normal cells treated with adrenaline?
  A. cells that lack adenylyl cyclase
  B. cells that lack the GPCR
  C. cells that lack cAMP phosphodiesterase
  D. cells that have an  subunit that cannot hydrolyze GTP but can interact properly with the  and  subunits
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Harley0507Harley0507
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alyssaa1017 Author
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6 years ago
Good timing, thanks!
elizabetheldrid
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Yesterday
Thanks
kevin12323h
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2 hours ago
This site is awesome
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