EDV in hemorrhaging

In cardiovascular physiology, end-diastolic volume (EDV) is the volume of blood in the right and/or left ventricle at end load or filling in (diastole). Because greater EDVs cause greater distention of the ventricle, EDV is often used synonymously with preload, which refers to the length of the sarcomeres in cardiac muscle prior to contraction (systole). An increase in EDV increases the preload on the heart and, through the Frank-Starling mechanism of the heart, increases the amount of blood ejected from the ventricle during systole (stroke volume).

Because nearly two-thirds of the blood in the systemic circulation is stored in the venous system, end-diastolic volume is closely related to venous compliance.

Increasing venous compliance elevates the capacitance of the veins, reducing venous return and therefore end-diastolic volume.

Decreasing venous compliance has the opposite effect. For example, activation of the baroreceptor reflex (occurring, for instance, in acute hemorrhage) causes vasoconstriction, which decreases venous compliance, improves venous return, and therefore increases end-diastolic volume.

end-diastolic volume (EDV) will increases

A person wit a massive loss of blood loses vital brain function which in turn can cause them to stop breathing. Also, loss of a limb could occur because of the lack of blood traveling throughout the body.