The human Prader–Willi and Angelman diseases result from

The human Prader–Willi and Angelman diseases result from
A.  mutated demethylase genes.
B.  deletion of a gene that controls X-inactivation.
C.  deletion of a region that controls gene imprinting.
D.  prions.
E.  gene duplication that causes overexpression of a methyltransferase.



The cellular effect of the abnormally oligomerized yeast protein Sup35 is
A.  failure of translation to terminate.
B.  abnormal chromosome inactivation by heterochromatin formation.
C.  abnormal imprinting of some genes.
D.  abnormal cytoskeletal structures.
E.  failure of chromosomes to condense during mitosis.


The mRNA of the Neurospora gene NMT1
A.  catalyzes a reaction involving its own product.
B.  base-pairs with other mRNAs to regulate their expression.
C.  has two domains; one is a coding sequence and the other is a ribozyme.
D.  is only transcribed when the substrate of its product is present.
E.  is alternatively spliced depending on the concentration of a metabolite of vitamin B1.