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SlideshowReport

Pathophysiology of septic shock.

Description
 

 

l

 

 

 

Endothelial cell damage
Activation of endothelial cells
Further release of endogenous mediators

Vasoconstricting mediators
(thromboxane, endothelin,
angiotensin II, and others)

 

 

 

 

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Vasodilating mediators
(prostanoids, nitric oxide)

 

 

Impaired vascular smooth
muscle tone

 

 

 

 

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