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13) In Drosophila, assume that the gene for scute bristles (s) is located at map position 0.0 and that the gene for ruby eyes (r) is at position 15.0. Both genes are located on the X chromosome and are recessive to their wild-type alleles. A cross is made between scute-bristled females and ruby-eyed males. Phenotypically wild F1 females were then mated to homozygous double mutant ma
The first genomic library ever made was for the beta-globin gene taken from human genomic dna of the reticulocytes. the library was done by using a partial digestion of the genome via restriction enzymes. [ a partial digest is done by not allowing the reaction to go to completion achieved by limiting incubation time] the fragments were then transferred to vectors of
The first genomic library ever made was for the beta-globin gene taken from human genomic dna of the reticulocytes. the library was done by using a partial digestion of the genome via restriction enzymes. [ a partial digest is done by not allowing the reaction to go to completion achieved by limiting incubation time] the fragments were then transferred to vectors of
Could someone please help me identify or look up or direct me towards finding the targets of this microRNA and its implications in breast cancer? hsa-miR-99 (human mature form)
Please help me answer this question. AaBbCc x aabbcc test cross with ABC linked and dominant. 2000 phenotypes observed with the following individuals counted out of 2000. establish the gene arrangement and map distances.
The binding of cAMP to the CAP protein involves numerous electrostatic interacti
The binding of cAMP to the CAP protein involves numerous electrostatic interactions. Most of the interactions are hydrogen bonds between cAMP atoms and the side chains of various amino acids in the CAP protein. Which of the following is a salt bridge rather than a hydrogen bond?
Complementation problem: possible region of deletion place the mutation on the thick line which ...
I need help with part B of this problem, I've asked Genetics tutors, other professors and even went on chegg to use the expert Q&A -- I've done part A and I know that 3 complements 1, 2 and 6 are unable to complement and 3,4,5 and 1,2 6 have similar mutations
but I have no idea if I am right? my genetics professor says that if I look at row 4 and
Wild type, or wt, refers to the genotype that is the typical genotype found in nature. Now read that again. Once more. Now answer. Answer
True False
I PCR out a mutated gene in a patient with nana chorea (the inability to dance in a coordinated fashion) and I compare it to another patient with that same disease phenotype.
Design a yeast strain that would turn blue when fed...... Please help!
1. Design a yeast strain that would turn blue when fed medium that contains hydrolyzed rice and beans (hydrolysis breaks proteins down to their constituent amino acids) plus threoninol and X-gal. Draw the structure of the gene/mRNA that will give the yeast this phenotype and describe the molecular events that lead to the yeast turning blue on medium containing hydrolyzed rice
Was Lewis just mapping them on a linkage map based on mutations?
Hi guys,
So I've spent a few days reviewing the genetic tools used in labs, but can't quite find the distinction between a few of the methods.
1. RT-PCR is used to determine if a specific gene is expressed; however, RNA-seq appears to do the same thing. I understand that RT-PCR uses a specific primer and thus tests the expression of one gene. Does RNA
Two different genes control the expression of coat colour in an organism.
3. Two different genes control the expression of coat colour in an organism. The allele B produces a black coat colour and the allele G produces a gray-striped coat. The gene B influences the expression of gene G. The gray-stripped coat pattern appears only when gene B is homozygous recessive. An albino (coat colour lacking) occurs when both genes are homozygous. A pure-breedin
Visualizing exons and their respective trancripts and protein domains.
I am building a script that takes exons from a particular gene and then plots them by their base pair coordinates on a 2D graph. Using data from Ensembl, I can match each exon to its respective transcript and protein domain.
I use data from Ensemble to match each domain to the gene base pairs responsible for its formation; I can then plot the protein domains above their r
1. The first child of this couple was Rh- (Rhesus negative). What is the chance that the second child of this is a girl and Rh- (Rhesus negative)? Use Punnett squares and/or the calculation to show your work.
2. Nomenclature for writing alleles on a homologous chromosome pair
The phrase DEP/dep means the first three letters, DEP, represent the order in
a. What is one possible double stranded DNA sequence you would insert into this gene is order to have it now make a protein with the 1o structure: NH2 MET ALA SER SER ILE VAL GLU COOH
b. Which bases in the default wildtype sequence (from # to #) would you replace with this new stretch of sequence?
im quite new to NCBI and genebank so heres my question
i need to extract sequences from 7 geneloci of 130 different bacterial strains deposited on genebank. those sequences were all submitted in the context of one single study. now instead of the time consuming process of extracting the sequences strain per strain in the taxonomy browser ,
Identify the most likely mode of inheritance found in this pedigree
I think that the most likely mode would be autosomal dominance with incomplete penetrance because it did skip one generation. Any help would be greatly appreciated. Let me know what you think it is and please also write why you think it is that. Thanks!
Thank you for reading. So I have a basic understanding of Mendel's works, but I am trying to figure genetics out better. Please if you could help, because I can't find the data anywhere on the internet on these questions.
If you take an F1 hybrid and cross it to a p1 (seperate from the hybrid's two different parent types) would you get a new F1 hybrid, or F
I am in serious need of correction or confirmation please! I am going to throw out some random facts and please let me know if I am accurate or not. Thanks for helping!
F1 hybrid can only be created by using two separate parents from inbred lines or P1. F1 are uniform, showing a new phenotype without variation. Not even once in a while will you see a second phenotype show
I got my finals in a few days and was wondering if anybody could give me the link to the answers to the review questions in this book? I finished all the questions but I'm not entirely sure how to check them as there is no answer section and the wwnorton.com/studyspace doesn't have anything either. Thanks
Assume that during meiosis I, none of the C chromosomes disjoin at metaphase, bu
Assume that during meiosis I, none of the C chromosomes disjoin at metaphase, but they separate into dyads (instead of monads) during meiosis II. Each resultant gamete participated in fertilization with a normal haploid gamete. What combinations will result?
Check all that apply. A) two copies of chromosome A, two copies of chromosome B, four copies of chromos
Recombination and the Role of Chromosomal Exchange
I read my genetic book for this, but I can't seem to find the answers. Please help if possible.
PART A The discernible difference in length between the two X chromosomes of the female fruit fly was important in Stern’s experiments because _______. A) it allowed cytological detection of physical exchange between the chromosomes B) it allowed him to p
someone put only the answers but would you put it the full answer please instead only putting A,B Thanks. Case Studies: Pediatrics: Congenital Heart Disease: Billy Adams
Fruit Fly genetics: WILD MALE x BROWN EYE DUMPY WING female
Question pertains to Pearson biologylabsonline homework. Assignment is to cross a wild male X brown eye, dumpy wing female. The F1 results were as follows: 100% wild F1 phenotype, autosomal results.
Backcrossed a F1 male to a P1 female. F2 Results were: 1:1 ratio of wild phenotypes to brown eye, dumpy wing. There were no flys showing wild eyes, du
A black body color (as opposed to the wild-type yellow color) in fruit flies is due to the recessive ebony allele, e. Small wings are due to the recessive vestigal allele, vg. You wish to deduce the genotype of a yellow-bodied large-winged male fruit fly. What are all the possible genotypes this male could be at the ebony and vestigal loci? What is the genotype an
In humans, a hypothetical trait is under the control of a single gene, A.
In humans, a hypothetical trait is under the control of a single gene, A. Four alleles have been found: A1, which is dominant to all others; A2, which is dominant to A3 and A4, which are codominant.
If gene A is on an autosome, how many different genotypes are possible?
If gene A is on an autosome, how many different phenotypes are possible?
The question asks to calculate the tandem repeats in the D1S80 allele. i plotted the graph but im not sure what to do from there. next question asks to be figure outhow many diffferent classes of homozygous and heterozygous exist? any help will be greatly appreciated
here is the data, i figured out that there is three overlaps in the Hind3 and EcoR1 at the 21200 and the 350 mark and the 2700 mark. that means it was cut twice? but after that i dont know where to go.
A mutation in which gene could be responsible for this biosynthetic auxotroph
One of my mutants [EMS-mutagenized, ampicillin enriched, 99.9% killing] grew only [! only] on a plate containing arginine, aspartate, proline, glutamic acid, inositol, and ornithine. I've spent hours trying to find out where the mutation might be [i have found out how all the amino acids on the plate are related using ecocyc], but i can't pinpoint an intermediate that woul
The table shown lists the approximate new mutation rates for three autosomal dom
The table shown lists the approximate new mutation rates for three autosomal dominant human diseases. Trait Mutations per 106 gametes Retinoblastoma (tumor of the retina) 20 Achondroplasia (statural dwarfism) 80 Neurofibromatosis (tumor of nervous tissue) 220
In a series of 50000 consecutive live births recorded in a
Can someone please tell me what is the formulae for calculating genetic map units?
Here is the question from my quiz:
The cross GE/ge × ge/ge produces the following progeny: GE/ge 404, ge/ge 396, gE/ge 97, Ge/ge 103. From these data, one can conclude that there are 20 map units between the G and E loci.
1. 12 out of 2400 babies that were born at a maternity hospital died shortly after birth from colonic obstruction due to a recessive allele. Determine the frequency of the lethal allele in the population, expressed as a percent.
2. 10 of 2400 babies born at a maternity hospital died shortly after birth from colonic obstruction due to a recessive allele. What percent
