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vanpac vanpac
wrote...
Posts: 612
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6 years ago
The most common portal of entry for bacteremia is:
 
  a. the genitourinary tract.
  b. the respiratory tract.
  c. an abscess.
  d. the biliary tract.



The laboratory method of choice to identify Mycoplasma and Ureaplasma infections is:
 
  a. enzyme-linked immunosorbent assay (ELISA).
  b. direct fluorescent antibody (DFA).
  c. latex agglutination.
  d. real-time polymerase chain reaction (PCR).



In the identification of mycobacterial isolates, the niacin test involves the:
 
  a. accumulation of niacin in the medium in which the organism is growing.
  b. metabolism of niacin to nicotinic acid by enzymatic action.
  c. testing of the isolate for susceptibility to niacin.
  d. utilization of niacin in metabolic pathways to produce nicotinic acid.



Which group of drugs is effective against M. pneumoniae infections?
 
  a. Lactams
  b. Sulfonamides
  c. Cephalosporins
  d. Fluoroquinolones



This flagellate is pear-shaped with falling leaf motility. It contains variant-specific surface proteins (VSPs) on its surface, which provide a mechanism for the organism to survive in the small intestine as a result of its ability to resist intestinal proteases. The organism is:
 
  a. G. duodenalis.
  b. C. mesnili.
  c. D. fragilis.
  d. E. hominis.
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Replies
wrote...
6 years ago
A
The most common portals of entry for bacteremia are the genitourinary tract (25), respiratory tract (20), abscesses (10), surgical wound infections (5), biliary tract (5), miscellaneous sites (10), and uncertain sites (25).

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D
Several amplification methods, including real-time PCR, have been developed for the detection of the clinically relevant Mycoplasma and Ureaplasma species.

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A
Niacin (nicotinic acid) plays an important role in the oxidation-reduction reactions that occur during mycobacterial metabolism. Niacin therefore accumulates in the medium in which these organisms are growing. A positive niacin test is preliminary evidence that the organism that ex-hibits a buff-colored, slow-growing, rough colony may be M. tuberculosis.

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D
Because of the lack of a cell wall, M. pneumoniae are innately resistant to all lactam antibiotics. In addition, they are resistant to sulfonamides, trimethoprim, and rifampin. Susceptibility patterns vary by species to macrolides and lincosamides. M. pneumoniae is usually susceptible to macrolides, tetracycline, ketolides, and fluoroquinolones.

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A
The VSPs are a family of related, highly unusual proteins covering the surface of the organism. The VSPs are resistant to the effects of intestinal proteases, thus allowing the parasites to survive in the protease-rich small intestine. An antigenic variation of the surface antigen during human infections with G. duodenalis has been documented.
vanpac Author
wrote...
6 years ago
Thank you soooo very much, was really helpful and kind of you to answer my q's
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