Activity 2 Q 3. Identify which of the stimulus modalities induced the largest amplitude receptor potential in the Pacinian corpuscle. How well did the results compare with your prediction?
A2 Q4. Identify which of the stimulus modalities induced the largest-amplitude recptor potential in the olfactory receptors. How well the the results compare with you prediction?
A2 Q 6. What type of sensory neuron would likely respond to a green light?
Activity 3 Q 2. What change in membrane potential (depolarization or hyperpolarization) triggers an action potential?
A3 Q 3. How did the action potential at R1 (or R2) change as you increased the stimulus voltage above the threshold voltage? How well did the results compare with your prediction?
A3 Q 5. What part of a neuron was investigated in this activity?
Activity 4 Q 4. Why are fewer action potentials recorded at R2 when TTX is applied between R1 and R2? How well did the results compare with you rprediction?
A4 Q 5. Why are fewer action potentials recorded at R2 when lidocaine is applied between R1 and R2? How well did the results compare with your prediction?
A4 Q 6. Pain-sensitive neurons (called nociceptors) conduct action potentials from the skin or teeth to sites in the brain involved in pain perception. Where should a dentist inject the lidocaine to block pain perception?
Activity 5 Q 2. Define the absolute refractory period.
A5 Q 3. How did the threshold for the second action potential change as you further decreased the interval between stimuli? How well did the results compare with your prediction?
A5 Q 4. What is it harder to generate a second action potential during the relative refractory period?
Activity 6 Q 1. Why are multiple action potentials generated in response to a long stimulus that is above threshold?
A6 Q 2. Why does the frequency of action potentials increase when the stimulus intensity increases? How well did the results compare with your prediction?
A6 Q 3. How does threshold change during the relative refractory period?
A6 Q 4. What is the relationship between the interspike interval and the frequency of action potentials?
Activity 7 Q 1. How did the conduction velocity in the B fiber compare with that in the A fiber? How well did the results compare with your prediction?
A7 Q 2. How did the conduction velocity in the C fiber compare with that in the B fiber? How well did the results compare with your prediction?
A7 Q 3. What is the effect of axon diameter on conduction velocity?
A7 Q 4. What is the effect of the amount of myelination on conduction velocity?
A7 Q 5. Why did the time between the stimulation and the action potential at R1 differ for each axon?
A7 Q 6. Why did you need to change the timescale on the oscilloscope for each axon?
Activity 8 Q 1. When the stimulus intensity is increased, what changes: the number of synaptic vesicles released or the amount of neurotransmitter per vesicle?
A8 Q 2. What happened to the amount of neurotransmitter release when you switched from the control extracellular fluid to the extracellular fluid with no Ca2+? How well did the results compare with your prediction?
A8 Q 3. What happened to the amount of neurotransmitter release when you switched from the extracellular fluid with no Ca2+ to the extracellular fluid with low Ca2+? How well did the results compare with your prediction?
A8 Q 4. How did neurotransmitter release in the Mg2+ extracellular fluid compare to that in the control extracellular fluid? How well did the result compare with your prediction?
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